New Studies on Myelin Repair for Multiple Sclerosis
Multiple sclerosis (MS) is an autoimmune disorder that takes place when the immune system attacks the central nervous system. The disease leads to demyelination – when the myelin sheath of neurons that wrap around and insulate the nerve fibers is destroyed. Demyelination causes impairment to cognition, movement and sensation. Although a lot is known about the disease process of multiple sclerosis, the cause remains unknown.
Scientists have been working on new treatments for multiple sclerosis, such as beta interferons, natalizumab, and glatiramer acetate. These treatments target the inflammation process by decreasing the inflammation that exists in early stages of the disease. Unfortunately, there are no effective treatments that target myelin, by protecting or repairing it, to help reduce or prevent nerve fiber loss due to demyelination.
The Edinburgh MS Research Center, founded by the MS Society of Scotland, is working to find a treatment for myelin repair. They are currently aware that myelin is damaged by MS in two ways – both the myelin and the cells that make myelin are attacked and destroyed, or the myelin is attacked and destroyed, but the cells that make myelin survive. They also know that the way myelin is damaged will be different from person to person, so the treatments will need to be different depending upon the processes.
This information has led them to believe that they need to either transplant or generate new cells for those who have experienced damage to both the myelin and the cells that make myelin. They believe this can be done by using the brain’s own stem cells to generate new cells. Due to MS being a disease which shows degeneration over time, the natural repair process can fail, therefore this hypothesis is complicated and will take much research, testing and studies to be fully developed.
For those who only experience the myelin being damaged but not the cells that make myelin, they are looking toward models of repair and reactivation.
The Edinburgh S Research Center in Scotland will be studying new theories, recruiting additional scientists and collaborating with other centers and scientists around the world to work diligently toward an understanding of myelin and myelin repair for patients with multiple sclerosis.
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Scientists have been working on new treatments for multiple sclerosis, such as beta interferons, natalizumab, and glatiramer acetate. These treatments target the inflammation process by decreasing the inflammation that exists in early stages of the disease. Unfortunately, there are no effective treatments that target myelin, by protecting or repairing it, to help reduce or prevent nerve fiber loss due to demyelination.
The Edinburgh MS Research Center, founded by the MS Society of Scotland, is working to find a treatment for myelin repair. They are currently aware that myelin is damaged by MS in two ways – both the myelin and the cells that make myelin are attacked and destroyed, or the myelin is attacked and destroyed, but the cells that make myelin survive. They also know that the way myelin is damaged will be different from person to person, so the treatments will need to be different depending upon the processes.
This information has led them to believe that they need to either transplant or generate new cells for those who have experienced damage to both the myelin and the cells that make myelin. They believe this can be done by using the brain’s own stem cells to generate new cells. Due to MS being a disease which shows degeneration over time, the natural repair process can fail, therefore this hypothesis is complicated and will take much research, testing and studies to be fully developed.
For those who only experience the myelin being damaged but not the cells that make myelin, they are looking toward models of repair and reactivation.
The Edinburgh S Research Center in Scotland will be studying new theories, recruiting additional scientists and collaborating with other centers and scientists around the world to work diligently toward an understanding of myelin and myelin repair for patients with multiple sclerosis.
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